http://heikebean.com/nutrition.htm
Ich richte mich nicht nach Blutbildern, weil ich die Aussagekraft für sehr gering bis missverständlich halte. Eine Erklärung zum Selen im Serum:
Hier ist noch etwas mehr zu Selen:Target Blood Selenium Levels
Whole blood selenium is the preferred test, since it measures both selenium in the plasma and
selenium inside red blood cells. With marginal intakes of selenium, the red cell levels will drop first,
so serum or plasma testing may show adequate levels when total body selenium is actually borderline
or low. For maximal antioxidant protection, the target level is 200 to 250 ug/L (micrograms per liter).
This target should be used for active horses. Levels of 160 to 180 ug/L may be adequate for inactive
horses. Serum or plasma levels will be around 25% lower than whole blood.
(Aus einer Vorlesung von Kellon- und ja, ich würde andere zitieren, nur gibt es soweit ich weiss von niemandem sonst bisher Informationen auf dem Niveau.)The site of selenium absorption in equine intestines has not been examined. In other species, absorption
occurs primarily in the small intestine. Selenium is readily absorbed, but with different efficiencies in
different studies, suggesting the potential for mineral interactions when using inorganic selenium
sources. This is entirely consistent with other species, where high copper intakes, or compounds such
as sulfur and arsenic in drinking water can interfere with inorganic selenium absorption. When feeding
copper at above the minimum requirements, and using inorganic selenium sources, it is wise to
also increase selenium intake. For example, if feeding 200% of the NRC for copper, do the same for
selenium.
Under conditions of unhindered absorption (no mineral competition), there is a safety factor of
approximately 10 fold between required level and one that may produce chronic toxic effects in other
species, including humans. The current NRC recommended intake is 0.1 ppm in the diet, or 1 mg/day
at maintenance for a 500 kg horse. However, they also recognized this may be too low for optimal
immune function. A 2001 study found better antibody production in foals from mares receiving 3 mg
Se per day rather than 1 mg.
Suggested intakes for exercising horses are only marginally higher than maintenance and still below 2
mg/day for a 500 kg horse, even with extremely heavy work loads. These recommendations do not take
into consideration that selenium is released from red blood cells during exercise, and selenium
excretion/loss in urine is higher after exercise:
http://www.ker.com/library/researchsummaries/sum05.pdf
These urinary losses may well be why many endurance riders swear by much higher selenium intakes
than are usually used – and do not have elevated blood seleniums on these high Se intakes.
Caution: If you are going to experiment with higher than recommended selenium intakes, do it safely.
Check the horse's whole blood selenium before you start and at 3 to 4 week intervals.
As mentioned earlier, using organic selenium source, selenium yeast, avoids problems with mineral
competition reducing absorption. However, it may leave much less wiggle room for experimenting with
dose before reaching toxicity both because it is more efficiently absorbed, and also better retained.
http://www.jvdi.org/cgi/reprint/18/1/61
Better retention was also documented in the KER study. A final consideration that needs more study is
how the chelated forms are distributed in the body tissues and metabolized. Selenium yeast contains
naturally formed chelated selenium in the form of selenocysteine so that shouldn't be a problem.
However, chelated Se in the form of L-selenomethionine is also available and in some supplements.
This may not behave the same way:
http://www.ncbi.nlm.nih.gov/pubmed/1803 ... rez.Pubmed.
Pubmed_ResultsPanel.Pubmed_RVDocSum
da ist das Orthocomplex nicht so viel teurer.
